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Bishop, J. M.
Bishop, J. Michael
Bishop, John Michael
Ioannes Michael Bishop
J. Michael Bishop (American immunologist and microbiologist)
J. Michael Bishop (amerykański immunolog i mikrobiolog, noblista)
John Michael Bishop
John Michael Bishop (Amerikaans bioloog)
John Michael Bishop (amerikai immunológus és virológus)
John Michael Bishop (biologo statunitense)
John Michael Bishop (US-amerikanischer Virologe)
Джон Майкл Бишоп (американский иммунолог и микробиолог)
Джон Майкл Бішоп
Жон Майкл Бишоп
جون مايكل بيشوب
جی. مایکل بیشاپ
جے مائیکل بشوپ
জন মাইকেল বিশপ
ビショップ, J. マイケル
Greaves, Melvyn F. (1941-)
Greaves, Melvyn Francis (1941-)
Rowley, Janet D.
Scientific American, Inc
Scientific American, Inc. (New York)
University of California, Los Angeles
Weinberg, Robert A. (1942-)
Weinberg, Robert Allan (1942-....))
Weinberg, Robert Allan (1942-)
Activation of the Met receptor by cell attachment induces and sustains hepatocellular carcinomas in transgenic mice.
Acute promyelocytic leukemia: from treatment to genetics and back
BCL-2 cooperates with promyelocytic leukemia retinoic acid receptor alpha chimeric protein (PMLRARalpha) to block neutrophil differentiation and initiate acute leukemia.
Beta PDGF receptor mutants defective for mitogenesis promote neurite outgrowth in PC12 cells.
c-Myc regulates mammalian body size by controlling cell number but not cell size.
Cell division and cell survival in the absence of survivin
Cellular adherence elicits ligand-independent activation of the Met cell-surface receptor.
Conditional transformation of cells and rapid activation of the mitogen-activated protein kinase cascade by an estradiol-dependent human raf-1 protein kinase.
Contrasting patterns of myc and N-myc expression during gastrulation of the mouse embryo.
Cre-mediated gene inactivation demonstrates that FGF8 is required for cell survival and patterning of the first branchial arch.
DFer gene of Drosophila melanogaster encodes two membrane-associated proteins that can both transform vertebrate cells, The
Directed establishment of rat brain cell lines with the phenotypic characteristics of type 1 astrocytes.
Distinct pathways of genomic progression to benign and malignant tumors of the liver.
dominant mutant allele of the ING4 tumor suppressor found in human cancer cells exacerbates MYC-initiated mouse mammary tumorigenesis., A
Drosophila myb is required for the G2/M transition and maintenance of diploidy.
Enteropathogenic E. coli acts through WASP and Arp2/3 complex to form actin pedestals.
Expression during Drosophila development of DER, a gene related to erbB-1 and neu: correlations with mutant phenotypes.
Expression of N-myc in teratocarcinoma stem cells and mouse embryos.
Farnesyl transferase inhibitors induce extended remissions in transgenic mice with mature B cell lymphomas.
Gan idenshi wa dokokara kitaka
gene related to the proto-oncogene fps/fes is expressed at diverse times during the life cycle of Drosophila melanogaster., A
Genes and cancer : proceedings of a CETUS-UCLA Symposium held in Steamboat Springs, Colorado, March 11-17, 1984
Genome-wide screen for allelic imbalance in a mouse model for neuroblastoma.
Genome-wide search for loss of heterozygosity in transgenic mouse tumors reveals candidate tumor suppressor genes on chromosomes 9 and 16.
Hedgehog elicits signal transduction by means of a large complex containing the kinesin-related protein costal2.
Hedgehog-stimulated phosphorylation of the kinesin-related protein Costal2 is mediated by the serine/threonine kinase fused.
How to win the Nobel Prize : an unexpected life in science
Id1 cooperates with oncogenic Ras to induce metastatic mammary carcinoma by subversion of the cellular senescence response
Identification of a large Myc-binding protein that contains RCC1-like repeats.
Inactivation of a c-Myb/estrogen receptor fusion protein in transformed primary cells leads to granulocyte/macrophage differentiation and down regulation of c-kit but not c-myc or cdc2
Inhibition of CDK1 as a potential therapy for tumors over-expressing MYC
Intranuclear localization of human papillomavirus 16 E7 during transformation and preferential binding of E7 to the Rb family member p130
Leukemia initiated by PMLRARalpha: the PML domain plays a critical role while retinoic acid-mediated transactivation is dispensable.
Localization of the human AHNAK/desmoyokin gene (AHNAK) to chromosome band 11q12 by somatic cell hybrid analysis and fluorescence in situ hybridization.
loxP-directed cloning: Use of Cre recombinase as a universal restriction enzyme
macrophage-stimulating protein pathway promotes metastasis in a mouse model for breast cancer and predicts poor prognosis in humans., The
Macrophages are required for cell death and tissue remodeling in the developing mouse eye.
Mary Lasker and Her Prizes: An Appreciation
MET and MYC cooperate in mammary tumorigenesis
MOLECULAR ANALYSIS OF TUMOR VIRUSES, THE
molecular genetics of cancer: 1988., The
Molecular themes in oncogenesis.
Mouse mammary tumor virus/v-Ha-ras transgene-induced mammary tumors exhibit strain-specific allelic loss on mouse chromosome 4
MYB and MYC in the cell cycle.
myb provides an essential function during Drosophila development.
Myristoylation and differential palmitoylation of the HCK protein-tyrosine kinases govern their attachment to membranes and association with caveolae
Negative control of the Myc protein by the stress-responsive kinase Pak2.
Neoplastic transformation by truncated alleles of human NOTCH1/TAN1 and NOTCH2.
Nerve growth factor rapidly stimulates tyrosine phosphorylation of phospholipase C-gamma 1 by a kinase activity associated with the product of the trk protooncogene.
Nestin-Cre transgenic mouse line Nes-Cre1 mediates highly efficient Cre/loxP mediated recombination in the nervous system, kidney, and somite-derived tissues
Neurotrophin-3 is required for proper cerebellar development.
new transgenic mouse line for tetracycline inducible transgene expression in mature melanocytes and the melanocyte stem cells using the Dopachrome tautomerase promoter, A
Overexpression of MYC causes p53-dependent G2 arrest of normal fibroblasts.
PEBP2 beta MYH11 fusion created by Inv(16)(p13; q22) in myeloid leukemia impairs neutrophil maturation and contributes to granulocytic dysplasia, The
PEBP2betaMYH11 fusion created by Inv(16)(p13;q22) in myeloid leukemia impairs neutrophil maturation and contributes to granulocytic dysplasia., The
Phosphorylation of the fused protein kinase in response to signaling from hedgehog.
PML-RARA-targeted DNA vaccine induces protective immunity in a mouse model of leukemia.
PMLRARalpha transgene initiates murine acute promyelocytic leukemia., A
Predisposition to neoplastic transformation caused by gene replacement of H-ras1.
Proto-oncogenes and plasticity in cell signaling.
protooncogene MYC can break B cell tolerance, The
Redefining the expression and function of the inhibitor of differentiation 1 in mammary gland development.
Retinoic Acid and Arsenic Synergize to Eradicate Leukemic Cells in a Mouse Model of Acute Promyelocytic Leukemia
Retroviruses and cancer genes.
Reversible tumorigenesis by MYC in hematopoietic lineages.
RNA interference (RNAi) with RNase III-prepared siRNAs.
ROLE OF VIRION-ASSOCIATED DNA POLYMERASE STUDY
ROUS SARCOMA VIRUS: REPLICATION AND CELL TRANSFORMATION
Scientific American molecular oncology
screen for genes that suppress loss of contact inhibition: Identification of ING4 as a candidate tumor suppressor gene in human cancer, A
Senescence of human fibroblasts induced by oncogenic Raf
Short RNA duplexes produced by hydrolysis with Escherichia coli RNase III mediate effective RNA interference in mammalian cells.
Src interacts with dynamin and synapsin in neuronal cells
STUDY OF THE ROLE OF VIRION-ASSOCIATED DNA POLYMERASE
Suppressor of Fused represses Gli-mediated transcription by recruiting the SAP18-mSin3 corepressor complex.
Sustained loss of a neoplastic phenotype by brief inactivation of MYC.
Targeted expression of MYCN causes neuroblastoma in transgenic mice
Telomerase extends the lifespan of virus-transformed human cells without net telomere lengthening.
Therapeutic potential of a synthetic lethal interaction between the MYC proto-oncogene and inhibition of aurora-B kinase
Torso, a receptor tyrosine kinase required for embryonic pattern formation, shares substrates with the sevenless and EGF-R pathways in Drosophila.
Transient excess of MYC activity can elicit genomic instability and tumorigenesis.
Contributed to or performed:
PROCEEDINGS- NATIONAL ACADEMY OF SCIENCES USA