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Person
ISNI: 
0000 0001 2412 2967
Name: 
Zholos, A.
Zholos, A V
Zholos, Alexander
Creation class: 
article
Text
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Creation role: 
author
Related names: 
Queen's University Belfast
Titles: 
Ca2+- and volume-sensitive chloride currents are differentially regulated by agonists and store-operated Ca2+ entry.
Ca2+-independent Phospholipase A2-dependent gating of TRPM8 by lysophospholipids.
Deletion of TRPC4 and TRPC6 in mice impairs smooth muscle contraction and intestinal motility in vivo.
Effects of polyamines on the muscarinic receptor-operated cation current in guinea-pig ileal smooth muscle myocytes.
G-protein-gated TRP-like cationic channel activated by muscarinic receptors: effect of potential on single-channel gating.
G-protein gated TRP-like cationic channel activated by muscarinic receptors: effects of potential on single channel gating
Invited review: Regulation of TRP-like muscarinic cation current in gastrointestinal smooth muscle with special reference to PLC-InsP sub(3)-Ca super(2+) system
Isoform-specific Inhibition of TRPC4 Channel by Phosphatidylinositol 4,5-Bisphosphate
Liquidity and price discovery on the London stock exchange
Muscarinic Cholinergic Excitation of Smooth Muscle: Signal Transduction and Single Cationic Channel Properties
Muscarinic receptor-activated cationic channels in murine ileal myocytes
Pharmacology of transient receptor potential melastatin channels in the vasculature.
Potential synergy: voltage-driven steps in receptor-G protein coupling and beyond.
Prostate cell differentiation status determines transient receptor potential melastatin member 8 channel subcellular localization and function.
Regulation of TRP-like muscarinic cation current in gastrointestinal smooth muscle with special reference to PLC/InsP3/Ca2+ system.
Rho kinase and protein kinase C involvement in vascular smooth muscle myofilament calcium sensitization in arteries from diabetic rats
TRPC7 Is a Receptor-Operated DAG-Activated Channel in Human Keratinocytes
TRPM channels in the vasculature.
Contributed to or performed: 
ACTA PHARMACOLOGICA SINICA
Notes: 
Sources: 
JNAM
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