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Person
ISNI: 
0000 0001 2417 8020
Name: 
Williams, A. C.
Williams, Ann
Creation class: 
Text
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Creation role: 
author
Related names: 
University of Bristol
Titles: 
acidic environment leads to p53 dependent induction of apoptosis in human adenoma and carcinoma cell lines: implications for clonal selection during colorectal carcinogenesis., An
BAG-1 is up-regulated in colorectal tumour progression and promotes colorectal tumour cell survival through increased NF- Kappa B activity
Direct Transcriptional Up-regulation of Cyclooxygenase-2 by Hypoxia-Inducible Factor (HIF)-1 Promotes Colorectal Tumor Cell Survival and Enhances HIF-1 Transcriptional Activity during Hypoxia
endogenous cannabinoid, anandamide, induces cell death in colorectal carcinoma cells: a possible role for cyclooxygenase 2., The
Genomic imprinting at the WT1 gene involves a novel coding transcript (AWT1) that shows deregulation in Wilms' tumours
Hedgehog signalling in colorectal tumour cells: Induction of apoptosis with cyclopamine treatment
importance of cyclic nucleotides (cyclic GMP and cyclic AMP) in the development of malignant disease, The
In vitro models for studying colorectal carcinogenesis: cellular and molecular events including APC and Rb cleavage in the control of proliferation, differentiation and apoptosis.
Increased EP4 Receptor Expression in Colorectal Cancer Progression Promotes Cell Growth and Anchorage Independence
Increased p53-dependent Apoptosis by the Insulin-like Growth Factor Binding Protein IGFBP-3 in Human Colonic Adenoma-derived Cells
Induction of apoptosis by the 16-kDa amino-terminal fragment of the insulin-like growth factor binding protein 3 in human colonic carcinoma cells
Insulin-like growth factor binding protein 3 (IGFBP-3) potentiates TRAIL-induced apoptosis of human colorectal carcinoma cells through inhibition of NF-kappaB.
Insulin-like growth factor binding protein (IGFBP-3) potentiates TRAIL-induced apoptosis of human colorectal carcinoma cells through inhibition of NF-κB
Interaction between [beta]-catenin and HIF-1 promotes cellular adaptation to hypoxia
Is the downregulation of EphB2 receptor expression during colorectal tumorigenesis due to hypoxia?
Nuclear BAG-1 expression inhibits apoptosis in colorectal adenoma-derived epithelial cells.
Prostaglandin F(2alpha) stimulates motility and invasion in colorectal tumor cells.
retinoblastoma protein interacts with Bag-1 in human colonic adenoma and carcinoma derived cell lines., The
role of the retinoblastoma protein (Rb) in the nuclear localization of BAG-1: implications for colorectal tumour cell survival, The
Subcellular localisation of BAG-1 and its regulation of vitamin D receptor-mediated transactivation and involucrin expression in oral keratinocytes: Implications for oral carcinogenesis
Transcriptional down-regulation of the retinoblastoma protein is associated with differentiation and apoptosis in human colorectal epithelial cells
Transcriptional upregulation of the insulin-like growth factor binding protein IGFBP-3 by sodium butyrate increases IGF-independent apoptosis in human colonic adenoma-derived epithelial cells
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